Vitamin B1 Capsules Thiamine REDWELLS No Additives High Strength 200mg - 60 Pack

Product Information

None. Unless you are deficient in thiamine, TTFD will almost certainly do more harm than good. Even if you are deficient in vitamin B1, there are much better supplemental forms. Thank you for your reply and advice. This particular doctor is a top Nephrologist here in NY and he’s also been a close friend since I was in my teens so there were a few advantages talking to him on the subject. In terms of alcohol consumption, about a year ago, for the first time in my life, I start chronic drinking. I stopped for 3 months after the Papilledema diagnosis, but then about 3.5 weeks ago started again, this time consuming much less alcohol. Around 2 weeks ago is when I noticed the memory and balance issue. I have stopped drinking again. Any light you could shed on this would be greatly appreciated.. Thanking you in advance for your time and help. Thiamine is found in health food stores as thiamine hydrochloride and thiamine mononitrate. These are known as “salts” of thiamine. Like dietary thiamine, they require a protein transporter to get the vitamin into the cell. Their absorption used to be thought to be extremely limited, but megadoses are effective in some situations. The absorption of salts is therefore inferior to that of the thiamine derivatives discussed above. They are all so-called “open ring (thiazole)” forms of thiamine and represent the most useful way of getting big doses of thiamine into the cell. The reader should be aware that when we talk about big doses of a vitamin, it is being used as a drug. Although they can be used for simple vitamin deficiency, their medical use goes far beyond that because they can be effective sometimes when thiamine absorption is genetically compromised. We Need Your Help

Sleep Problems: TTDF has increase my energy and increased rapid/random thought and I wake early (-50%) A few of us were familiar with your work from perhaps 10 years ago, and several experimented with Benfotiamine at that time. While some saw some good gains from that, I feel that my son (now in his 20s) is seeing spectacular gains from the allithiamine after only a week or two–a much more obvious response than we saw with regular B vitamins or the Benfotiamine. Thiamine, also known as vitamin B1, exists in a variety of thiamine-rich food sources including beef, liver, dried milk, oats, pork, eggs, seeds, legumes, peas and fortified grain and it is vitally important to maintain normal body function. Thiamine could play an important role in the nervous system [ 1, 2], heart health [ 3], energy metabolism [ 4], and psychological health [ 5]. A recommended dietary allowance (RDA) has been announced by the U.S. Food and Nutrition Board for vitamin B1 (thiamin) to prevent the deficiency in a healthy population (Food and Nutrition Board). The RDA could be calculated by calorie intake referring to 0.5 mg per 1000 kcal and the intake range could be 1.0–1.1 mg per day for women and 1.2–1.5 mg for men, referring to an average caloric intake. The specific population also showed inadequate dietary intake and thiamin insufficiency so the RDA could be increased to 1.5 mg/day for health maintenance [ 6]. Physical activity could also affect the thiamine requirement in addition to gender, age, and physiological status [ 7]. Thiamine could be converted to three phosphorylated forms, thiamine monophosphate (TMP), thiamine pyrophosphate (TPP), and thiamine triphosphate (TTP), after intestinal absorption, but the rate-limiting transport system for thiamine absorption causes a bioavailability issue [ 8]. Why might I keep experiencing these “crashes” after exertion if my body is supplied with enough thaimine? B12-responsive Methylmalonic acidaemia: Genetic defect encoding the methylmalonyl-CoA mutase enzyme, causing low affinity for adenosylcobalamin cofactor and a pathological accumulation of methylmalonic acid. This condition can be treated with megadoses of B12.cases of sudden-onset deafness was treated with 150mg TTFD orally, with most therapeutic effect seen after 2-3 months of treatment I researched my problem continuously, trying supplements and elimination diets. I found things that helped: GFCF diet, Ceylon cinnamon, raising my vit D and magnesium levels, sublingual B12, methylfolate (could dampen tingles if I took a super high dose), potassium chloride. I tried literally dozens of things.

I am experiencing an electrolyte imbalance (low phosphorus and potassium) after a period of fasting during Lent. I began eating normally again and experienced what I originally thought was Refeeding Syndrome. I had been diagnosed with this in the past and like this time, despite treatment and supplementing with up to 3,000 mg of Phosphorus and 160 mEq of Potassium, my blood levels are still low.

You may also like

I do not currently take any prescription medications although the MD’s want me to take Toprol XL and Lexapro and Progesterone cream to manage the symptoms of dysautonomia and perimenopause. I do take a large quantity of supplements. Perhaps one of the below affects thiamine absorption? Prosultiamine (TPD) at 300mg per day for 12 weeks (TPD, a very similar molecule to TTFD) used to treat spinal cord injury in human T lymphotropic virus type I-associated myelopathy/tropical spastic paraparesis (2013). Significant improvement in motor functions and bladder control, as well as reducing viral numbers in blood. Only adverse symptom was mild epigastric discomfort. No safety concerns. Many thanks for your speedy reply – it’s very much appreciated! We have corresponded before regarding Lupron and the truly appalling effects it has on the body.

Note – I have not yet felt benefit from this regimen for my longstanding Fluoroqunolone antibiotic toxicity characterized by peripheral neuropathy and musculoskeletal pain or longstanding anxiety/OCD/insomnia. The reason for me wanting to try Lipotiamine, is because it should be able to bypass the stomach, and get absorbed much better then conventional b1, and bypass issues like thiaminase. Or I have understood it wrong, but I take your word for it, that it is the best b1 to take. The strange thing is that my body reacts even to artificial sweeteners and stuff like alluose, which your body doesn’t even really absorb. It makes me question if there’s something wrong in my bowels and there’s bad bacteria feeding on stuff I put down there. Maybe my past chronic alcohol use, my IBS has a role in it, I have my ups and downs, but I just can’t handle the stress of my memory anymore, and I am only 33. I do know that I was feeling a lot better on the Lipothiamine and wondering whats the best course now in terms of supplementation given my situation.

I suffered a disc rupture (L5/s1) needing surgery 27.12.17 as it had sequestered. Ouch!! Pain relief was instant but I didn’t make a good recovery with on going burning stinging nerve pain. I used to be a heavy weekend drinker, have had high stress jobs (sister in a NICU in the UK) and life stress losing both parents before they aged 70, mum to breast cancer aged 62 and dad to general ill health resulting in an above knee amputation. In the 8 year period they passed I also had a baby. I am wondering if there’s a genetic component to my probable thiamine deficiency? I eat a pretty basic diet of lean animal protein, vegetables and fruits, olive and coconut oil, with occasional sprouted grain and cheddar cheese, 2-3 liters of water per day with LMNT electrolytes. Benfotiamine is an S-acyl derivative of thiamine known for its ability to improve blood sugar management and support peripheral nerve function.

As a kid I was very active, tanned easily, had a lot of energy and pretty normal as far as kids go. In my country, prescribing antibiotics is normal enough for general problems (even when it has no effect), not sure it’s related but I thought worth mentioning as it’d have been prescribed to me a few times when growing up. Around the age of 12, I started to have issues with my gut. I would get intense abdominal pain and stopped going to school as a result. Shortly after this, I became very pale and very weak physically, I was far weaker than anyone else in the school by a significant margin and so pale that it everyone noticed and used to insult me over it. The changes were clearly very extreme and abnormal. Over the years after this, I gained weight and continued to be relatively physically weak and very pale (unable to tan). It stayed like this relatively unchanged until I hit my 20s. Worth noting, my diet as a kid was quite bad, it consisted of a lot of junk food and soda, as I was very fussy with food. It was difficult to get me to eat meat/vegetables and I never consumed fruit. This was less a case during my teenage years and not at all into my 20s. Also worth noting, once the stomach issues began, I became heavily constipated and had consistent issues with digestion ever since, particularly it seems with stuff that are very “doughy”, like pizza would have a high chance of triggering abdominal pain. At some point in this timeline, I was also prescribed PPIs which I used for over a month for acid reflux issues (PPIs are horrendous of course, I know that now). If it is W.E. is it still possible to reverse or mitigate/halt the memory/cognitive deterioration using the protocol two weeks after the symptoms started? (which is about now) From what I’ve since read, I realise this is likely paradox symptoms. I’m now on btwen 62.5-125mg (1/4 – 1/2 tab) daily, and I find I still have to take the dose at night. Daytime doses make me too tired. Physiological fatigue is caused by inadequate rest, physical effort or mental strain and could be categorized as peripheral fatigue, produced by changes at or distal to the neuromuscular junction. Central fatigue originates at the central nervous system (CNS), which decreases the neural drive to muscles. Fatigue development could result from different factors including metabolites, energy demands, oxygen availability, ion regulation, neural contributions, and physiological reactants during contractive processes [ 9]. Thiamin and folic acid deficiency accompanied by resistant electrolyte imbalance in the re-feeding syndrome in an elderly patient”I’v not been taking Lipothiamine for a little over a month. I believe the main symptoms of “paradox” have resolved which were emotional volatility, extra physical anxiety and chest pains. I have now increased the dosage to 200mg lipothiamine per day split in the following manner: 100mg at breakfast, 50mg at lunch, 50mg at dinner. Continuing to take the Alithiamine, after a few weeks, my stools just became what I would call “perfect” bowel movements, consistently. I was astonished. I also tended to feel a bit more energy and have not really felt any “air hunger” to an extreme level in quite a while. I continued taking it for a few months at the same 1 capsule 50mg dose until then I stopped taking Alithiamine for a little while to see what would happen and I noticed a slight increase in “air hunger” and constipation after a week or so. So I started taking it again and I noticed the symptoms decreased again. I am looking for the possibility of being able to do the ETK test to confirm a diagnosis but it is difficult here, so it may not be possible. I’ve been having problems waking up with a dry mouth for years, and recently air hunger, I thought was due to apena, however ENT could not diagnose me with sleep apnea after reading sleep study. He could not figure out way I was suffocating at night as he never had a skinny patient before. I found your article about pusedo-hypoxia after I talked to the ENT. subjects without nutritional deficiency, 20 cases of alcoholism, and 48 cases of alcoholism with signs of deficiency and/or liver disease were given either TTFD, Thiamine propyldisulfide (a similar disulfide derivative), or thiamine HCL. T hey showed no toxic effects at 3-6 months in any group, and demonstrated that oral TTFD/TPD increased whole blood, erythrocyte, and cerebrospinal fluid thiamine levels at an equivalent level to intravenous thiamine HCL. cases of herpes zoster treated with TTFD in conjunction with acyclovir and traditional Chinese medicine (2013).